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Clec2d Joins the Cell Death Sensor Ranks

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dc.contributor.authordel Fresno, Carlos
dc.contributor.authorSancho, David
dc.contributor.funderAsociación Española Contra el Cáncer
dc.contributor.funderUnión Europea. Comisión Europea. European Research Council (ERC)
dc.contributor.funderUnión Europea. Comisión Europea
dc.contributor.funderMinisterio de Ciencia, Innovación y Universidades (España)
dc.contributor.funderUnión Europea. Fondo Europeo de Desarrollo Regional (FEDER/ERDF)
dc.contributor.funderInstituto de Salud Carlos III
dc.contributor.funderFondation ACTERIA (Acting on European Research in Immunology and Allergology)
dc.contributor.funderComunidad de Madrid (España)
dc.contributor.funderAtresmedia
dc.contributor.funderFundación La Marató TV3
dc.contributor.funderFundación ProCNIC
dc.date.accessioned2020-04-20T07:40:49Z
dc.date.available2020-04-20T07:40:49Z
dc.date.issued2020-01
dc.description.abstractSensing tissue damage is an ancient function of immune cells that is central to the regulation of inflammation, tissue repair, and immunity. In this issue of Immunity, Lai et al. (2020) uncover a role for the C-type lectin receptor Clec2d as a sensor of cell death, which directly detects histones released during necrosis and thus contributes to inflammation and immunopathology.es_ES
dc.description.sponsorshipC.d.F. is supported by the AECC Foundation (INVES192DELF). Work in the DS laboratory is funded by the CNIC; the European Research Council (ERC 2016 Consolidator Grant 725091); the European Commission (635122-PROCROP H2020); Ministerio de Ciencia, Innovacion e Universidades (MICINN), Agencia Estatal de Investigacion, and Fondo Europeo de Desarrollo Regional (FEDER) (SAF2016-79040-R); Comunidad de Madrid (B2017/BMD-3733 Immunothercan-CM); FIS-Instituto de Salud Carlos III, MICINN, and FEDER (RD16/0015/0018-REEM); the Acteria Foundation; Atresmedia (Constantes y Vitales prize); and Fundacio La Marato de TV3 (201723). The CNIC is supported by the Instituto de Salud Carlos III (ISCIII), the MICINN, and the Pro CNIC Foundation and is a Severo Ochoa Center of Excellence (SEV-2015-0505).es_ES
dc.embargo.terms2021-01-01es_ES
dc.format.number1es_ES
dc.format.page6-8es_ES
dc.format.volume52es_ES
dc.identifier.citationImmunity. 2020; 52(1):6-8es_ES
dc.identifier.doi10.1016/j.immuni.2019.12.015es_ES
dc.identifier.e-issn1097-4180es_ES
dc.identifier.issn1074-7613es_ES
dc.identifier.journalImmunityes_ES
dc.identifier.pubmedID31951550es_ES
dc.identifier.urihttp://hdl.handle.net/20.500.12105/9619
dc.language.isoenges_ES
dc.publisherCell Presses_ES
dc.relation.projectIDinfo:eu-repo/grantAgreement/ES/SEV-2015-0505es_ES
dc.relation.projectIDinfo:eu-repo/grantAgreement/EC/H2020/725091es_ES
dc.relation.projectIDinfo:eu-repo/grantAgreement/EC/H2020/635122es_ES
dc.relation.projectIDinfo:eu-repo/grantAgreement/ES/SAF2016-79040-Res_ES
dc.relation.publisherversionhttps://doi.org/10.1016/j.immuni.2019.12.015es_ES
dc.repisalud.institucionCNICes_ES
dc.repisalud.orgCNICCNIC::Grupos de investigación::Inmunobiologíaes_ES
dc.rights.accessRightsopen accesses_ES
dc.rights.licenseAttribution-NonCommercial-NoDerivatives 4.0 Internacional*
dc.rights.urihttp://creativecommons.org/licenses/by-nc-nd/4.0/*
dc.subject.meshCell Deathes_ES
dc.subject.meshHumanses_ES
dc.subject.meshInflammationes_ES
dc.subject.meshNecrosises_ES
dc.subject.meshReceptors, Cell Surfacees_ES
dc.subject.meshHistoneses_ES
dc.subject.meshLectins, C-Typees_ES
dc.titleClec2d Joins the Cell Death Sensor Rankses_ES
dc.typejournal articlees_ES
dc.type.hasVersionAMes_ES
dspace.entity.typePublication
relation.isAuthorOfPublicationc56ae6c1-121b-4e6c-ab1c-ae787f8622f2
relation.isAuthorOfPublication58aa2591-8084-4500-bfe4-8f2c54e398e9
relation.isAuthorOfPublication.latestForDiscoveryc56ae6c1-121b-4e6c-ab1c-ae787f8622f2

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