Martinez-Val, ALynch, CJCalvo, IXimĂ©nez-EmbĂșn, PGarcia, FZarzuela, ESerrano, MMunoz, J2026-02-252026-02-252021-03-25Nat Commun. 2021 Mar 25;12(1):1863.https://hdl.handle.net/20.500.12105/27272We thank all members of the Proteomics Unit (CNIO), the Cellular Plasticity and Disease Group (IRB) and A. Efeyan (CNIO) for discussions. We thank O. Fernandez-Capetillo and M. Drosten for antibodies. We also thank the CNIO Experimental Therapeutics Programme for work on the CDK8/19i. I.C. is a recipient of predoctoral research contract from AGAUR. Work in the laboratory of M.S. was funded by the IRB and by the Spanish Ministry of Science co-funded by the European Regional Development Fund (ERDF) (SAF2017-82613-R), the European Research Council (ERC-2014-AdG/669622), and "laCaixa" Foundation. This work was supported by J.M. grant SAF2013-45504-R (MINECO). The CNIO Proteomics Unit belongs to ProteoRed, PRB3- ISCIII, supported by grant PT17/0019/0005. J.M. is supported by the Ramon y Cajal Programme (MINECO) RYC- 2012-10651. A.M.-V. is supported by BES-2014-070098 (MINECO).engVoRDissection of two routes to naive pluripotency using different kinase inhibitorsNATURE COMMUNICATIONSopen access