TY  - JOUR
AU  - Brandt, Marta
AU  - Grazioso, Tatiana P
AU  - Fawal, Mohamad-Ali
AU  - Tummala, Krishna S
AU  - Torres-Ruiz, Raul
AU  - Rodriguez Perales, Sandra
AU  - Perna, Cristian
AU  - Djouder, Nabil
PY  - 2018
DO  - 10.1016/j.cmet.2017.11.006
UR  - http://hdl.handle.net/20.500.12105/17554
AB  - Dietary habits that can induce inflammatory bowel disease (IBD) are major colorectal cancer (CRC) risk factors, but mechanisms linking nutrients, IBD, and CRC are unknown. Using human data and mouse models, we show that mTORC1 inactivation-induced...
LA  - eng
PB  - Cell Press
KW  - Adenoma
KW  - Adenomatous Polyposis Coli Protein
KW  - Carcinogenesis
KW  - Cell Proliferation
KW  - Chromosomal Instability
KW  - Colitis
KW  - Colorectal Neoplasms
KW  - DNA Damage
KW  - Female
KW  - HCT116 Cells
KW  - Homeostasis
KW  - Humans
KW  - Inflammation
KW  - Inflammatory Bowel Diseases
KW  - Interleukin-6
KW  - Intestines
KW  - Male
KW  - Mechanistic Target of Rapamycin Complex 1
KW  - Nuclear Proteins
KW  - RNA-Binding Proteins
KW  - Regeneration
KW  - Signal Transduction
KW  - Tumor Suppressor Protein p53
TI  - mTORC1 Inactivation Promotes Colitis-Induced Colorectal Cancer but Protects from APC Loss-Dependent Tumorigenesis.
TY  - journal article
ER  -