TY  - JOUR
AU  - Alonso-Herranz, Laura
AU  - Sahun-Español, Alvaro
AU  - Paredes, Ana
AU  - Gonzalo, Pilar
AU  - Gkontra, Polyxeni
AU  - Nunez, Vanessa
AU  - Clemente, Cristina
AU  - Cedenilla, Marta
AU  - Villalba-Orero, Maria
AU  - Inserte, Javier
AU  - Garcia-Dorado, David
AU  - Arroyo, Alicia G
AU  - Ricote, Mercedes
PY  - 2020
DO  - 10.7554/eLife.57920
UR  - http://hdl.handle.net/20.500.12105/13182
AB  - Macrophages (Mφs) produce factors that participate in cardiac repair and remodeling after myocardial infarction (MI); however, how these factors crosstalk with other cell types mediating repair is not fully understood. Here we demonstrated that...
LA  - eng
PB  - eLife Sciences Publications
KW  - Epithelial-Mesenchymal Transition
KW  - Animals
KW  - Collagen
KW  - Disease Models, Animal
KW  - Endothelium, Vascular
KW  - Female
KW  - Fibrosis
KW  - Flow Cytometry
KW  - Gene Expression Regulation
KW  - HEK293 Cells
KW  - Humans
KW  - Macrophages
KW  - Male
KW  - Matrix Metalloproteinase 14
KW  - Mice
KW  - Mice, Inbred C57BL
KW  - Mice, Knockout
KW  - Microcirculation
KW  - Myocardial Infarction
KW  - Phenotype
KW  - Reperfusion Injury
KW  - Transforming Growth Factor beta1
KW  - Ventricular Dysfunction, Left
TI  - Macrophages promote endothelial-to-mesenchymal transition via MT1-MMP/TGFβ1 after myocardial infarction.
TY  - journal article
ER  -