TY  - GEN
AU  - Blanco, Eduardo
AU  - Pavón, Francisco J
AU  - Palomino, Ana
AU  - Luque-Rojas, María Jesús
AU  - Serrano, Antonia
AU  - Rivera, Patricia
AU  - Bilbao, Ainhoa
AU  - Alen, Francisco
AU  - Vida, Margarita
AU  - Suáez, Juan
AU  - Rodríguez de Fonseca, Fernando
PY  - 2015
DO  - 10.1093/ijnp/pyu024
SN  - 1461-1457
UR  - http://hdl.handle.net/20.500.12105/18885
AB  - Background: Endocannabinoids modulate the glutamatergic excitatory transmission by acting as retrograde messengers. A growing body of studies has reported that both signaling systems in the mesocorticolimbic neural circuitry are involved in the...
LA  - eng
PB  - Oxford University Press
KW  - Cannabinoid
KW  - Cocaine
KW  - Sensitization
KW  - Glutamate
KW  - Prefrontal cortex
KW  - Cocaína
KW  - Inhibidores de la captación de dopamina
KW  - Discinesia inducida por medicamentos
KW  - Corteza prefrontal
KW  - ARN mensajero
KW  - Receptor cannabinoide CB1
KW  - Receptores de glutamato
KW  - Dopamine Uptake Inhibitors
KW  - Animals
KW  - Cocaine
KW  - Dopamine Uptake Inhibitors
KW  - Dyskinesia, Drug-Induced
KW  - Endocannabinoids
KW  - Glutamic Acid
KW  - Glutaminase
KW  - Lipoprotein Lipase
KW  - Male
KW  - Mice, Inbred C57BL
KW  - Monoacylglycerol Lipases
KW  - Prefrontal Cortex
KW  - RNA, Messenger
KW  - Receptor, Cannabinoid, CB1
KW  - Receptors, Glutamate
KW  - Amidohydrolases
TI  - Cocaine-induced behavioral sensitization is associated with changes in the expression of endocannabinoid and glutamatergic signaling systems in the mouse prefrontal cortex
TY  - research article
ER  -