TY  - JOUR
AU  - Tarín, Carlos
AU  - Fernández-Laso, Valvanera
AU  - Sastre, Cristina
AU  - Madrigal-Matute, Julio
AU  - Gómez, Mónica
AU  - Zaragoza, Carlos
AU  - Egido, Jesús
AU  - Burkly, Linda C
AU  - Martín-Ventura, Jose L
AU  - Blanco-Colio, Luis M
PY  - 2014
DO  - 10.1161/JAHA.113.000723
SN  - 2047-9980
UR  - http://hdl.handle.net/20.500.12105/7345
AB  - BACKGROUND: Abdominal aortic aneurysm (AAA) involves leukocyte recruitment, inflammatory cytokine production, vascular cell apoptosis, neovascularization, and vascular remodeling, all of which contribute to aortic dilatation. Tumor necrosis...
LA  - eng
PB  - American Heart Association (AHA)
KW  - Fn14
KW  - MMP activity
KW  - TWEAK
KW  - Aneurysm
KW  - Inflammation
KW  - Animals
KW  - Aortic Aneurysm, Abdominal
KW  - Apoptosis
KW  - Chemokine CCL2
KW  - Chemokine CCL5
KW  - Cytokine TWEAK
KW  - Disease Models, Animal
KW  - Inflammation
KW  - Macrophages
KW  - Matrix Metalloproteinase 9
KW  - Mice
KW  - Mice, Knockout
KW  - Muscle, Smooth, Vascular
KW  - Myocytes, Smooth Muscle
KW  - Neutrophils
KW  - Pancreatic Elastase
KW  - Receptors, Tumor Necrosis Factor
KW  - T-Lymphocytes
KW  - TWEAK Receptor
KW  - Tumor Necrosis Factors
TI  - Tumor necrosis factor-like weak inducer of apoptosis or Fn14 deficiency reduce elastase perfusion-induced aortic abdominal aneurysm in mice
TY  - journal article
ER  -