TY  - JOUR
AU  - Yoshida, Yohko
AU  - Shimizu, Ippei
AU  - Shimada, Atsuhiro
AU  - Nakahara, Keita
AU  - Yanagisawa, Sachiko
AU  - Kubo, Minoru
AU  - Fukuda, Shinji
AU  - Ishii, Chiharu
AU  - Yamamoto, Hiromitsu
AU  - Ishikawa, Takamasa
AU  - Kano, Kuniyuki
AU  - Aoki, Junken
AU  - Katsuumi, Goro
AU  - Suda, Masayoshi
AU  - Ozaki, Kazuyuki
AU  - Yoshida, Yutaka
AU  - Okuda, Shujiro
AU  - Ohta, Shigeo
AU  - Okamoto, Shiki
AU  - Minokoshi, Yasuhiko
AU  - Oda, Kanako
AU  - Sasaoka, Toshikuni
AU  - Abe, Manabu
AU  - Sakimura, Kenji
AU  - Kubota, Yoshiaki
AU  - Yoshimura, Norihiko
AU  - Kajimura, Shingo
AU  - Zuriaga, Maria
AU  - Walsh, Kenneth
AU  - Soga, Tomoyoshi
AU  - Minamino, Tohru
PY  - 2022
DO  - 10.1038/s41598-022-19245-x
UR  - http://hdl.handle.net/20.500.12105/15759
AB  - Low body temperature predicts a poor outcome in patients with heart failure, but the underlying pathological mechanisms and implications are largely unknown. Brown adipose tissue (BAT) was initially characterised as a thermogenic organ, and recent...
LA  - eng
PB  - Nature Publishing Group
KW  - Heart Failure
KW  - Myocardial Infarction
KW  - Adipocytes, Brown
KW  - Adipose Tissue, Brown
KW  - Animals
KW  - Choline
KW  - Methylamines
KW  - Mice
KW  - Thermogenesis
TI  - Brown adipose tissue dysfunction promotes heart failure via a trimethylamine N-oxide-dependent mechanism.
TY  - journal article
ER  -