TY  - GEN
AU  - Rivera, Patricia
AU  - Bindila, Laura
AU  - Pastor, Antoni
AU  - Pérez-Martín, Margarita
AU  - Pavón, Francisco-Javier
AU  - Serrano, Antonia
AU  - de la Torre, Rafael
AU  - Lutz, Beat
AU  - Rodríguez de Fonseca, Fernando
AU  - Suárez, Juan
PY  - 2015
DO  - 10.3389/fncel.2015.00098
UR  - http://hdl.handle.net/20.500.12105/17086
AB  - Endocannabinoids participate in the control of neurogenesis, neural cell death and gliosis. The pharmacological effect of the fatty acid amide hydrolase (FAAH) inhibitor URB597, which limits the endocannabinoid degradation, was investigated in the...
LA  - eng
PB  - Frontiers Media
KW  - URB597
KW  - FAAH
KW  - Cannabinoids
KW  - Energy metabolism
KW  - Neurogenesis
KW  - Gliosis
KW  - Bromodesoxiuridina
KW  - Cannabinoides
KW  - Carbamatos
KW  - Caspasa 3
KW  - Muerte celular
KW  - Proliferación celular
KW  - Colesterol
KW  - Endocannabinoides
KW  - Etanolaminas
KW  - Glucosa
KW  - Hipocampo
KW  - Hipotálamo
KW  - Microglía
KW  - Ácidos oléicos
KW  - Ácidos palmíticos
KW  - Ratas
KW  - Receptor cannabinoide CB1
KW  - Triglicéridos
KW  - Amidohidrolasas
KW  - Apoptosis
KW  - Astrocitos
KW  - Benzamidas
KW  - Peso corporal
KW  - Cerebro
KW  - Amidohydrolases
KW  - Animals
KW  - Apoptosis
KW  - Astrocytes
KW  - Benzamides
KW  - Body Weight
KW  - Brain
KW  - Bromodeoxyuridine
KW  - Cannabinoids
KW  - Carbamates
KW  - Caspases
KW  - Cell Death
KW  - Cell Proliferation
KW  - Cholesterol
KW  - Endocannabinoids
KW  - Ethanolamines
KW  - Gliosis
KW  - Glucose
KW  - Hippocampus
KW  - Hypothalamus
KW  - Microglia
KW  - Neurogenesis
KW  - Oleic Acids
KW  - Palmitic Acids
KW  - Rats
KW  - Receptor, Cannabinoid, CB1
KW  - Triglycerides
TI  - Pharmacological blockade of the fatty acid amide hydrolase (FAAH) alters neural proliferation, apoptosis and gliosis in the rat hippocampus, hypothalamus and striatum in a negative energy context.
TY  - research article
ER  -