TY  - JOUR
AU  - Cuadrado, Myriam
AU  - Martinez-Pastor, Barbara
AU  - Murga, Matilde
AU  - Toledo, Luis I
AU  - Gutierrez-Martinez, Paula
AU  - Lopez, Eva
AU  - Fernandez-Capetillo, Oscar
PY  - 2006
DO  - 10.1084/jem.20051923
SN  - 0022-1007
UR  - http://hdl.handle.net/20.500.12105/17688
AB  - DNA double-strand breaks (DSBs) are among the most deleterious lesions that can challenge genomic integrity. Concomitant to the repair of the breaks, a rapid signaling cascade must be coordinated at the lesion site that leads to the activation of cell...
LA  - eng
PB  - Rockefeller University Press
KW  - Ataxia Telangiectasia
KW  - Ataxia Telangiectasia Mutated Proteins
KW  - Cell Cycle
KW  - Cell Cycle Proteins
KW  - Cell Line, Transformed
KW  - Cell Line, Tumor
KW  - Checkpoint Kinase 1
KW  - Checkpoint Kinase 2
KW  - Chromatin
KW  - Chromosome Breakage
KW  - DNA Damage
KW  - DNA Replication
KW  - DNA-Binding Proteins
KW  - Flow Cytometry
KW  - Gamma Rays
KW  - Humans
KW  - Phosphorylation
KW  - Protein Kinases
KW  - Protein Serine-Threonine Kinases
KW  - Signal Transduction
KW  - Tumor Suppressor Proteins
TI  - ATM regulates ATR chromatin loading in response to DNA double-strand breaks.
TY  - journal article
ER  -