TY  - GEN
AU  - Pros, E
AU  - Saigi, M
AU  - Alameda, D
AU  - Gomez-Mariano, Gema Maria
AU  - Martinez-Delgado, Beatriz
AU  - Alburquerque-Bejar, J J
AU  - Carretero, J
AU  - Tonda, R
AU  - Esteve-Codina, A
AU  - Catala, I
AU  - Palmero, R
AU  - Jove, M
AU  - Lazaro, C
AU  - Patiño-García, Ana
AU  - Gil-Bazo, I
AU  - Verdura, S
AU  - Teulé, A
AU  - Torres-Lanzas, J
AU  - Sidransky, D
AU  - Reguart, N
AU  - Pio, R
AU  - Juan-Vidal, O
AU  - Nadal, E
AU  - Felip, E
AU  - Montuenga, L M
AU  - Sanchez-Cespedes, M
PY  - 2020
DO  - 10.1016/j.annonc.2019.09.001
UR  - http://hdl.handle.net/20.500.12105/17266
AB  - Background: The etiology and the molecular basis of lung adenocarcinomas (LuADs) in nonsmokers are currently unknown. Furthermore, the scarcity of available primary cultures continues to hamper our biological understanding of non-smoking-related lung...
LA  - eng
PB  - Elsevier
KW  - Nonsmokers
KW  - Lung adenocarcinoma
KW  - EGFR
KW  - RB1
KW  - Tyrosine kinase inhibitors
KW  - Whole-exome sequencing
KW  - ErbB Receptors
KW  - Lung Neoplasms
KW  - Adenocarcinoma of Lung
KW  - Drug Resistance, Neoplasm
KW  - Humans
KW  - Microtubule-Associated Proteins
KW  - Mutation
KW  - Protein Kinase Inhibitors
KW  - Retinoblastoma Binding Proteins
KW  - Ubiquitin-Protein Ligases
TI  - Genome-wide profiling of non-smoking-related lung cancer cells reveals common RB1 rearrangements associated with histopathologic transformation in EGFR-mutant tumors
TY  - research article
ER  -