TY  - GEN
AU  - Rodriguez-Cuenca, Sergio
AU  - Carobbio, Stefania
AU  - Barcelo-Coblijn, Gwendolyn
AU  - Prieur, Xavier
AU  - Relat, Joana
AU  - Amat, Ramon
AU  - Campbell, Mark
AU  - Dias, Ana Rita
AU  - Bahri, Myriam
AU  - Gray, Sarah L
AU  - Vidal-Puig, Antonio
PY  - 2018
DO  - 10.1111/dom.13370
SN  - 1462-8902
UR  - https://hdl.handle.net/20.500.12105/22618
AB  - Aims: Familial partial lipodystrophic syndrome 3 (FPLD3) is associated with mutations in the transcription factor PPAR gamma. One of these mutations, the P467L, confers a dominant negative effect. We and others have previously investigated the...
LA  - eng
PB  - Wiley
KW  - fatty liver
KW  - fibrates
KW  - lipodystrophy
KW  - P465L-PPAR gamma
KW  - PPAR alpha
KW  - Hyperlipidemias
KW  - PPAR gamma
KW  - Mice, Transgenic
KW  - Hypolipidemic Agents
KW  - Leucine
KW  - Mutation, Missense
KW  - Fatty Liver
KW  - Disease Models, Animal
KW  - Drug Resistance
KW  - Animals
KW  - Amino Acid Substitution
KW  - Proline
KW  - Fibric Acids
KW  - Mice
TI  - P465L-PPAR gamma mutation confers partial resistance to the hypolipidaemic action of fibrates
TY  - research article
ER  -