TY  - JOUR
AU  - Shao, Lijian
AU  - Sottoriva, Kilian
AU  - Palasiewicz, Karol
AU  - Zhang, Jizhou
AU  - Hyun, James
AU  - Soni, Sweta S
AU  - Paik, Na Yoon
AU  - Gao, Xiaopei
AU  - Cuervo, Henar
AU  - Malik, Asrar B
AU  - Rehman, Jalees
AU  - Lucas, Daniel
AU  - Pajcini, Kostandin V
PY  - 2019
DO  - 10.3324/haematol.2018.208660
UR  - http://hdl.handle.net/20.500.12105/17208
AB  - Loss-of-function studies have determined that Notch signaling is essential for hematopoietic and endothelial development. By deleting a single allele of the Notch1 transcriptional activation domain we generated viable, post-natal mice exhibiting...
LA  - eng
PB  - Fondazione Ferrata Storti
KW  - Cellular Microenvironment
KW  - Regeneration
KW  - Signal Transduction
KW  - Animals
KW  - Bone Marrow
KW  - Endothelial Cells
KW  - Fluorouracil
KW  - Gamma Rays
KW  - Gene Expression Profiling
KW  - Gene Expression Regulation
KW  - Hematopoietic Stem Cells
KW  - Humans
KW  - Mice
KW  - Mice, Knockout
KW  - Pancytopenia
KW  - Receptor, Notch1
KW  - Receptor, TIE-2
TI  - A Tie2-Notch1 signaling axis regulates regeneration of the endothelial bone marrow niche.
TY  - journal article
ER  -