TY  - GEN
AU  - Amorós-Pérez, Marta
AU  - Zuriaga, María A
AU  - Zorita, Virginia
AU  - Ramos-Neble, Beatriz L
AU  - Matesanz, Nuria
AU  - Plötner, Clara
AU  - Di Carli, Milena
AU  - Mol, Eline L C
AU  - Moro, Rosa
AU  - Cuevas, Diego
AU  - Andrés, Vicente
AU  - Fuster, José J
PY  - 2026
DO  - 10.1161/ATVBAHA.125.323734
UR  - https://hdl.handle.net/20.500.12105/27750
AB  - BACKGROUND: Clonal hematopoiesis driven by somatic mutations is an emerging cardiovascular risk factor, and the DNA damage response gene is among the most frequently mutated genes. Mutations in are enriched in cancer patients and survivors, where...
LA  - eng
PB  - Lippincott Williams & Wilkins (LWW)
KW  - atherosclerosis
KW  - cardiovascular diseases
KW  - clonal hematopoiesis
KW  - macrophages
KW  - radiation
KW  - Animals
KW  - Disease Models, Animal
KW  - Bone Marrow Transplantation
KW  - Mice, Inbred C57BL
KW  - Phenotype
KW  - Receptors, LDL
KW  - Mice, Knockout
KW  - Plaque, Atherosclerotic
KW  - Macrophages
KW  - Male
KW  - Genetic Predisposition to Disease
KW  - Cell Proliferation
KW  - Mice
TI  - No Effect of -Mutant Clonal Hematopoiesis on Atherosclerosis Development in Mice.
TY  - research article
ER  -