TY  - JOUR
AU  - Quintana-Cabrera, Ruben
AU  - Quirin, Charlotte
AU  - Glytsou, Christina
AU  - Corrado, Mauro
AU  - Urbani, Andrea
AU  - Pellattiero, Anna
AU  - Calvo, Enrique
AU  - Vazquez, Jesus
AU  - Enriquez, Jose Antonio
AU  - Gerle, Christoph
AU  - Soriano, Maria Eugenia
AU  - Bernardi, Paolo
AU  - Scorrano, Luca
PY  - 2018
DO  - 10.1038/s41467-018-05655-x
SN  - 2041-1723
UR  - http://hdl.handle.net/20.500.12105/6537
AB  - It is unclear how the mitochondrial fusion protein Optic atrophy 1 (OPA1), which inhibits cristae remodeling, protects from mitochondrial dysfunction. Here we identify the mitochondrial F1Fo-ATP synthase as the effector of OPA1 in mitochondrial...
LA  - eng
PB  - Nature Publishing Group
KW  - PERMEABILITY TRANSITION PORE
KW  - COMPLEX III DEFICIENCY
KW  - CYTOCHROME-C RELEASE
KW  - OXIDATIVE-PHOSPHORYLATION
KW  - F1F0-ATP SYNTHASE
KW  - SUPRAMOLECULAR ORGANIZATION
KW  - LIVER-MITOCHONDRIA
KW  - INHIBITOR PROTEIN
KW  - INNER MEMBRANE
KW  - SUBUNIT-G
TI  - The cristae modulator Optic atrophy 1 requires mitochondrial ATP synthase oligomers to safeguard mitochondrial function
TY  - journal article
ER  -