TY  - JOUR
AU  - Ruiz-Andres, Olga
AU  - Suarez-Alvarez, Beatriz
AU  - Sánchez-Ramos, Cristina
AU  - Monsalve, Maria
AU  - Sanchez-Niño, Maria D
AU  - Ruiz-Ortega, Marta
AU  - Egido, Jesús
AU  - Ortiz, Alberto
AU  - Sanz, Ana B
PY  - 2016
DO  - 10.1038/ki.2015.332
UR  - http://hdl.handle.net/20.500.12105/17391
AB  - Studies of mitochondria-targeted nephroprotective agents suggest a key role of mitochondrial injury in AKI. Here we tested whether an improved perception of factors responsible for mitochondrial biogenesis may provide clues to novel therapeutic...
LA  - eng
PB  - Elsevier
KW  - Acute Kidney Injury
KW  - Animals
KW  - Cells, Cultured
KW  - Cytokine TWEAK
KW  - Down-Regulation
KW  - Epigenesis, Genetic
KW  - Female
KW  - Histone Deacetylases
KW  - Kidney
KW  - Mice, Inbred C57BL
KW  - Mitochondria
KW  - NF-kappa B
KW  - Organelle Biogenesis
KW  - Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha
KW  - Receptors, Tumor Necrosis Factor
KW  - TWEAK Receptor
KW  - Tumor Necrosis Factors
TI  - The inflammatory cytokine TWEAK decreases PGC-1α expression and mitochondrial function in acute kidney injury.
TY  - journal article
ER  -