TY  - GEN
AU  - Pintado-Berninches, Laura
AU  - Fernandez-Varas, Beatriz
AU  - Benitez-Buelga, Carlos
AU  - Manguan-Garcia, Cristina
AU  - Serrano-Benitez, Almudena
AU  - Iarriccio, Laura
AU  - Carrillo, Jaime
AU  - Guenechea, Guillermo
AU  - Egusquiaguirre, Susana P
AU  - Pedraz, Jose-Luis
AU  - Hernández, Rosa M
AU  - Igartua, Manoli
AU  - Arias-Salgado, Elena G
AU  - Cortes-Ledesma, Felipe
AU  - Sastre, Leandro
AU  - Perona, Rosario
AU  - Pintado-Berninches, Laura
AU  - Fernandez-Varas, Beatriz
AU  - Benitez-Buelga, Carlos
AU  - Manguan-Garcia, Cristina
AU  - Serrano-Benitez, Almudena
AU  - Iarriccio, Laura
AU  - Carrillo, Jaime
AU  - Guenechea, Guillermo
AU  - Egusquiaguirre, Susana P
AU  - Pedraz, Jose-Luis
AU  - Hernández, Rosa M
AU  - Igartua, Manoli
AU  - Arias-Salgado, Elena G
AU  - Sastre, Leandro
AU  - Perona, Rosario
PY  - 2019
UR  - https://hdl.handle.net/20.500.12105/25360
AB  - Ataxia telangiectasia (AT) is a genetic disease caused by mutations in the ATM gene but the mechanisms underlying AT are not completely understood. Key functions of the ATM protein are to sense and regulate cellular redox status and to transduce DNA...
LA  - eng
PB  - Nature Publishing Group
KW  - LINKED DYSKERATOSIS-CONGENITA
KW  - NEURAL STEM-CELLS
KW  - DNA-DAMAGE
KW  - CELLULAR-RESPONSE
KW  - ATM
KW  - STRESS
KW  - ACTIVATION
KW  - SENESCENCE
KW  - GENE
KW  - PATHOGENESIS
TI  - GSE4 peptide suppresses oxidative and telomere deficiencies in ataxia telangiectasia patient cells.
TY  - research article
ER  -