2024-03-29T08:33:06Zhttp://repisalud.isciii.es/oai/requestoai:repisalud.isciii.es:20.500.12105/96192023-10-13T07:05:50Zcom_20.500.12105_2145com_20.500.12105_2051com_20.500.12105_2144col_20.500.12105_2146
Repisalud
author
del Fresno, Carlos
author
Sancho, David
funder
Asociación Española Contra el Cáncer
funder
Unión Europea. Comisión Europea. European Research Council (ERC)
funder
Unión Europea. Comisión Europea
funder
Ministerio de Ciencia, Innovación y Universidades (España)
funder
Unión Europea. Fondo Europeo de Desarrollo Regional (FEDER/ERDF)
funder
Instituto de Salud Carlos III
funder
Fondation ACTERIA (Acting on European Research in Immunology and Allergology)
funder
Comunidad de Madrid (España)
funder
Atresmedia
funder
Fundación La Marató TV3
funder
Fundación ProCNIC
2020-04-20T07:40:49Z
2020-04-20T07:40:49Z
2020-01
Immunity. 2020; 52(1):6-8
1074-7613
http://hdl.handle.net/20.500.12105/9619
31951550
10.1016/j.immuni.2019.12.015
1097-4180
Immunity
Sensing tissue damage is an ancient function of immune cells that is central to the regulation of inflammation, tissue repair, and immunity. In this issue of Immunity, Lai et al. (2020) uncover a role for the C-type lectin receptor Clec2d as a sensor of cell death, which directly detects histones released during necrosis and thus contributes to inflammation and immunopathology.
eng
Clec2d Joins the Cell Death Sensor Ranks
journal article
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URL
https://repisalud.isciii.es/bitstream/20.500.12105/9619/1/Clec2dJoinsCellDeath_2020.pdf
File
MD5
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Clec2dJoinsCellDeath_2020.pdf
URL
https://repisalud.isciii.es/bitstream/20.500.12105/9619/3/Clec2dJoinsCellDeath_2020.pdf.txt
File
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Clec2dJoinsCellDeath_2020.pdf.txt