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dc.contributor.authordel Fresno, Carlos 
dc.contributor.authorSancho, David 
dc.date.accessioned2020-04-20T07:40:49Z
dc.date.available2020-04-20T07:40:49Z
dc.date.issued2020-01
dc.identifier.citationImmunity. 2020; 52(1):6-8es_ES
dc.identifier.issn1074-7613es_ES
dc.identifier.urihttp://hdl.handle.net/20.500.12105/9619
dc.description.abstractSensing tissue damage is an ancient function of immune cells that is central to the regulation of inflammation, tissue repair, and immunity. In this issue of Immunity, Lai et al. (2020) uncover a role for the C-type lectin receptor Clec2d as a sensor of cell death, which directly detects histones released during necrosis and thus contributes to inflammation and immunopathology.es_ES
dc.description.sponsorshipC.d.F. is supported by the AECC Foundation (INVES192DELF). Work in the DS laboratory is funded by the CNIC; the European Research Council (ERC 2016 Consolidator Grant 725091); the European Commission (635122-PROCROP H2020); Ministerio de Ciencia, Innovacion e Universidades (MICINN), Agencia Estatal de Investigacion, and Fondo Europeo de Desarrollo Regional (FEDER) (SAF2016-79040-R); Comunidad de Madrid (B2017/BMD-3733 Immunothercan-CM); FIS-Instituto de Salud Carlos III, MICINN, and FEDER (RD16/0015/0018-REEM); the Acteria Foundation; Atresmedia (Constantes y Vitales prize); and Fundacio La Marato de TV3 (201723). The CNIC is supported by the Instituto de Salud Carlos III (ISCIII), the MICINN, and the Pro CNIC Foundation and is a Severo Ochoa Center of Excellence (SEV-2015-0505).es_ES
dc.language.isoenges_ES
dc.publisherCell Presses_ES
dc.relation.isversionofPostprintes_ES
dc.rights.urihttp://creativecommons.org/licenses/by-nc-nd/4.0/*
dc.subject.meshCell Death es_ES
dc.subject.meshHumans es_ES
dc.subject.meshInflammation es_ES
dc.subject.meshNecrosis es_ES
dc.subject.meshReceptors, Cell Surfacees_ES
dc.subject.meshHistones es_ES
dc.subject.meshLectins, C-Type es_ES
dc.titleClec2d Joins the Cell Death Sensor Rankses_ES
dc.typeArtículoes_ES
dc.rights.licenseAttribution-NonCommercial-NoDerivatives 4.0 Internacional*
dc.identifier.pubmedID31951550es_ES
dc.format.volume52es_ES
dc.format.number1es_ES
dc.format.page6-8es_ES
dc.identifier.doi10.1016/j.immuni.2019.12.015es_ES
dc.contributor.funderFundación Científica AECCes_ES
dc.contributor.funderEuropean Research Counciles_ES
dc.contributor.funderEuropean Commissiones_ES
dc.contributor.funderMinisterio de Ciencia, Innovación y Universidades (MCNU)es_ES
dc.contributor.funderEuropean Regional Development Fund (ERDF/FEDER)es_ES
dc.contributor.funderInstituto de Salud Carlos III - ISCIIIes_ES
dc.contributor.funderFondation Acteriaes_ES
dc.contributor.funderComunidad de Madrides_ES
dc.contributor.funderAtresmedia Corporación de medios de comunicación Atreses_ES
dc.contributor.funderFundacio la Maratóes_ES
dc.contributor.funderFundación ProCNICes_ES
dc.embargo.terms2021-01-01es_ES
dc.identifier.e-issn1097-4180es_ES
dc.relation.publisherversionhttps://doi.org/10.1016/j.immuni.2019.12.015es_ES
dc.identifier.journalImmunityes_ES
dc.repisalud.orgCNICCNIC::Grupos de investigación::Inmunobiologíaes_ES
dc.repisalud.institucionCNICes_ES
dc.relation.projectIDinfo:eu-repo/grantAgreement/ES/SEV-2015-0505es_ES
dc.relation.projectIDinfo:eu-repo/grantAgreement/EC/H2020/725091es_ES
dc.relation.projectIDinfo:eu-repo/grantAgreement/EC/H2020/635122es_ES
dc.relation.projectIDinfo:eu-repo/grantAgreement/ES/SAF2016-79040-Res_ES
dc.rights.accessRightsinfo:eu-repo/semantics/embargoedAccesses_ES


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Attribution-NonCommercial-NoDerivatives 4.0 Internacional
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