A mouse model for Li-Fraumeni-Like Syndrome with cardiac angiosarcomas associated to POT1 mutations.

Mice with Pulmonary Fibrosis Driven by Telomere Dysfunction.

Short and dysfunctional telomeres protect from allergen-induced airway inflammation.

Telomerase deficiency and dysfunctional telomeres in the lung tumor microenvironment impair tumor progression in NSCLC mouse models and patient-derived xenografts.

Telomerase treatment prevents lung profibrotic pathologies associated with physiological aging.