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dc.contributor.authorBhaskaran, Abhishek
dc.contributor.authorFitzgerald, John
dc.contributor.authorJackson, Nicholas
dc.contributor.authorGizurarson, Sigfus
dc.contributor.authorNanthakumar, Kumaraswamy
dc.contributor.authorPorta-Sanchez, Andreu 
dc.date.accessioned2021-05-05T07:08:17Z
dc.date.available2021-05-05T07:08:17Z
dc.date.issued2020-12
dc.identifier.citationArrhythm Electrophysiol Rev. 2020; 9(4):211-218es_ES
dc.identifier.issn2050-3369es_ES
dc.identifier.urihttp://hdl.handle.net/20.500.12105/12868
dc.description.abstractEmpirical approaches to targeting the ventricular tachycardia (VT) substrate include mapping of late potentials, local abnormal electrogram, pace-mapping and homogenisation of the abnormal signals. These approaches do not try to differentiate between the passive or active role of local signals as the critical components of the VT circuit. By not considering the functional components, these approaches often view the substrate as a fixed anatomical barrier. Strategies to improve the success of VT ablation need to include the identification of critical functional substrate. Decrement-evoked potential (DeEP) mapping has been developed to elucidate this using an extra-stimulus added to a pacing drive train. With knowledge translation in mind, the authors detail the evolution of the DeEP concept by way of a study of simultaneous panoramic endocardial mapping in VT ablation; an in silico modelling study to demonstrate the factors influencing DeEPs; a multicentre VT ablation validation study; a practical approach to DeEP mapping; the potential utility of DeEPs to identify arrhythmogenic atrial substrate; and, finally, other functional mapping strategies.es_ES
dc.language.isoenges_ES
dc.publisherRadcliffe Medical Mediaes_ES
dc.relation.isversionofPublisher's versiones_ES
dc.rights.urihttp://creativecommons.org/licenses/by-nc/4.0/*
dc.titleDecrement Evoked Potential Mapping to Guide Ventricular Tachycardia Ablation: Elucidating the Functional Substrate.es_ES
dc.typeArtículoes_ES
dc.rights.licenseAtribución-NoComercial 4.0 Internacional*
dc.identifier.pubmedID33437489es_ES
dc.format.volume9es_ES
dc.format.number4es_ES
dc.format.page211-218es_ES
dc.identifier.doi10.15420/aer.2020.25es_ES
dc.description.peerreviewedes_ES
dc.relation.publisherversionhttps://doi.org/10.15420/aer.2020.25es_ES
dc.identifier.journalArrhythmia & electrophysiology reviewes_ES
dc.repisalud.orgCNICCNIC::Grupos de investigación::Cardiología Moleculares_ES
dc.repisalud.institucionCNICes_ES
dc.rights.accessRightsinfo:eu-repo/semantics/openAccesses_ES


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Atribución-NoComercial 4.0 Internacional
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