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ATP-dependent Lon protease controls tumor bioenergetics by reprogramming mitochondrial activity
Ischaemic conditioning and targeting reperfusion injury: a 30 year voyage of discovery
KrasP34R and KrasT58I mutations induce distinct RASopathy phenotypes in mice.
NF-kB signaling in cardiomyocytes is inhibited by sevoflurane and promoted by propofol.