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A common minimal motif for the ligands of HLA-B*27 class I molecules
A viral, transporter associated with antigen processing (TAP)-independent, high affinity ligand with alternative interactions endogenously presented by the nonclassical human leukocyte antigen E class I molecule
Antigen processing of a short viral antigen by proteasomes
Caspases in virus-infected cells contribute to recognition by CD8+ T lymphocytes
CD69 does not affect the extent of T cell priming
Complex antigen presentation pathway for an HLA-A*0201-restricted epitope from Chikungunya 6K protein
Computational characterization of the peptidome in transporter associated with antigen processing (TAP)-deficient cells
Conventional CD4+ T cells present bacterial antigens to induce cytotoxic and memory CD8+ T cell responses
Diversity of natural self-derived ligands presented by different HLA class I molecules in transporter antigen processing-deficient cells
Exogenous, TAP-independent lysosomal presentation of a respiratory syncytial virus CTL epitope.
H-2Ld class I molecule protects an HIV N-extended epitope from in vitro trimming by endoplasmic reticulum aminopeptidase associated with antigen processing.
HIV envelope protein inhibits MHC class I presentation of a cytomegalovirus protective epitope.
Mouse DC-SIGN/CD209a as Target for Antigen Delivery and Adaptive Immunity
Multiple proteases process viral antigens for presentation by MHC class I molecules to CD8(+) T lymphocytes.
Multiple viral ligands naturally presented by different class I molecules in transporter antigen processing-deficient vaccinia virus-infected cells
Need for tripeptidyl-peptidase II in major histocompatibility complex class I viral antigen processing when proteasomes are detrimental
Selective involvement of proteasomes and cysteine proteases in MHC class I antigen presentation.
Sequential cleavage by metallopeptidases and proteasomes is involved in processing HIV-1 ENV epitope for endogenous MHC class I antigen presentation.
Structural and Nonstructural Viral Proteins Are Targets of T-Helper Immune Response against Human Respiratory Syncytial Virus
The DC receptor DNGR-1 mediates cross-priming of CTLs during vaccinia virus infection in mice