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BPTF is required for c-MYC transcriptional activity and in vivo tumorigenesis
CREBBP/EP300 bromodomains are critical to sustain the GATA1/MYC regulatory axis in proliferation
In vivo inhibition of c-MYC in myeloid cells impairs tumor-associated macrophage maturation and pro-tumoral activities
Myc is dispensable for cardiomyocyte development but rescues Mycn-deficient hearts through functional replacement and cell competition.
ODZ1 allows glioblastoma to sustain invasiveness through a Myc-dependent transcriptional upregulation of RhoA