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A viral, transporter associated with antigen processing (TAP)-independent, high affinity ligand with alternative interactions endogenously presented by the nonclassical human leukocyte antigen E class I molecule
CD69 does not affect the extent of T cell priming
CD69 Targeting Enhances Anti-vaccinia Virus Immunity.
Multiple viral ligands naturally presented by different class I molecules in transporter antigen processing-deficient vaccinia virus-infected cells
The DC receptor DNGR-1 mediates cross-priming of CTLs during vaccinia virus infection in mice