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Nanog at the exit of pluripotency: new roles in the gastulating mouse embryo
Nanog regulates Pou3f1 expression at the exit from pluripotency during gastrulation
Our First Choice: Cellular and Genetic Underpinnings of Trophectoderm Identity and Differentiation in the Mammalian Embryo.
The pluripotency factor NANOG controls primitive hematopoiesis and directly regulates Tal1
Transient fibrosis resolves via fibroblast inactivation in the regenerating zebrafish heart
Transitions in cell potency during early mouse development are driven by Notch